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Making Spinal Cord Injury (SCI) Research Accessible to Everyone. Simplified summaries of the latest research, designed for patients, caregivers and anybody who's interested.

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Immunology Research

Browse the latest research summaries in the field of immunology for spinal cord injury patients and caregivers.

Showing 501-510 of 730 results

Regenerative MedicineImmunologyNeurology

Repurposing the cardiac glycoside digoxin to stimulate myelin regeneration in chemically-induced and immune-mediated mouse models of multiple sclerosis

Glia, 2022 • October 1, 2022

This study demonstrates the capacity of digoxin to induce OPC differentiation in vitro, to robustly stimulate myelination in vivo in the chemically-induced cuprizone and lysophosphatidylcholine (LPC) ...

KEY FINDING: Digoxin regulated multiple genes in oligodendrocyte progenitor cells (OPCs) essential for oligodendrocyte (OL) differentiation in vitro, promoted OL differentiation both in vitro and in vivo in female naïve C57BL/6J (B6) mice, and stimulated recovery of myelinated axons in B6 mice following demyelination in the corpus callosum induced by cuprizone and spinal cord demyelination induced by lysophosphatidylcholine (LPC), respectively.

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Spinal Cord InjuryImmunologyNeurology

Microglia coordinate cellular interactions during spinal cord repair in mice

Nature Communications, 2022 • July 21, 2022

This study demonstrates that microglia are vital for SCI recovery and coordinate injury responses in CNS-resident glia and infiltrating leukocytes. The research identifies specific signaling axes depe...

KEY FINDING: Microglia depletion exacerbates tissue damage and worsens functional recovery after SCI in mice.

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Regenerative MedicineImmunologyGenetics

Granulin as an important immune molecule involved in lamprey tissue repair and regeneration by promoting cell proliferation and migration

Cellular & Molecular Biology Letters, 2022 • September 2, 2022

This study investigates the evolutionary dynamics and function of progranulins (PGRNs) in lampreys, primitive vertebrates known for their regenerative abilities. Four genes encoding PGRNs were identif...

KEY FINDING: Four PGRN genes were identified in lampreys: one long form (Lr-PGRN-L) and three short forms (Lr-PGRN-S1, Lr-PGRN-S2, and Lr-PGRN-S3).

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COVID-19ImmunologyBiomedical

Nanoantidote for repression of acidosis pH promoting COVID-19 infection

VIEW, 2022 • March 2, 2022

This study reveals that acidosis-related pH promotes SARS-CoV-2 infection by increasing ACE2 expression on the cell membrane, suggesting a positive feedback loop where SARS-CoV-2 infection-induced aci...

KEY FINDING: Acidosis-related pH (6.8) increases the expression of SARS-CoV-2 receptor ACE2 on the cell membrane.

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Regenerative MedicineImmunologyDermatology

Tissue resident macrophages specifically express Lactotransferrin and Vegfc during ear pinna regeneration in spiny mice

Dev Cell, 2024 • February 26, 2024

The study dissects macrophage phenotypes in spiny mice (Acomys spp.) that regenerate ear pinnae tissue versus lab mice (Mus musculus) that form scar tissue, identifying secreted factors from activated...

KEY FINDING: Acomys macrophages exhibit a muted inflammatory profile compared to Mus macrophages and secrete factors that antagonize collagen production and drive matrix remodeling.

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ImmunologyNeurology

The antimicrobial peptide cathelicidin drives development of experimental autoimmune encephalomyelitis in mice by affecting Th17 differentiation

PLoS Biology, 2022 • August 26, 2022

This study demonstrates that the antimicrobial peptide cathelicidin drives severe autoimmune disease in the mouse model of MS. Exposure of CD4+ T cells in the draining lymph node to neutrophil-derived...

KEY FINDING: Cathelicidin is expressed by multiple cell types in lymphoid organs and the central nervous system during EAE, including neutrophils, endothelial cells and microglia/macrophages.

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ImmunologyNeurology

Secondary White Matter Injury Mediated by Neuroinflammation after Intracerebral Hemorrhage and Promising Therapeutic Strategies of Targeting the NLRP3 Inflammasome

Current Neuropharmacology, 2023 • January 1, 2023

This review summarizes the mechanisms by which neuroinflammation, mediated by the NLRP3 inflammasome, exacerbates secondary white matter injury (WMI) after intracerebral hemorrhage (ICH). The review d...

KEY FINDING: White matter injury (WMI) significantly contributes to neurological dysfunction after intracerebral hemorrhage (ICH), often more than previously recognized gray matter injuries.

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Spinal Cord InjuryImmunologyBioinformatics

Identification of hub genes related to the innate immune response activated during spinal cord injury

FEBS Open Bio, 2022 • January 1, 2022

This study identifies differentially-expressed innate immune-related genes and hub genes following spinal cord injury (SCI). Temporal expression patterns of these genes were analyzed and validated to ...

KEY FINDING: Nine hub genes (Ccl2, Stat3, Mapk14, Stat1, Tlr2, Cxcl10, Myd88, Itgam, and Mapk8) were identified as key players in the innate immune response after SCI.

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ImmunologyNeurologyBioinformatics

Cross‑regional homeostatic and reactive glial signatures in multiple sclerosis

Acta Neuropathologica, 2022 • September 16, 2022

The study generated an integrated cell type-specific transcriptomic atlas of MS pathology spanning three major CNS sites including leukocortical, cerebellar and spinal cord areas. The study identified...

KEY FINDING: While we found strong cross-regional diversity among glial subtypes in control tissue, regional signatures become more obscure in MS.

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ImmunologyNeurology

Transcriptomic Profiling Identifies CD8+ T Cells in the Brain of Aged and Alzheimer’s Disease Transgenic Mice as Tissue-Resident Memory T Cells

The Journal of Immunology, 2022 • September 15, 2022

The study explores the transcriptomic profile of CD8+ T cells in the brains of aged and Alzheimer's disease (AD) transgenic mice, comparing them to CD8+ T cells in the blood and identifying them as ti...

KEY FINDING: Brain CD8+ T cells in both AD transgenic and aged wild-type mice exhibit a gene signature characteristic of tissue-resident memory (Trm) T cells.

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