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Making Spinal Cord Injury (SCI) Research Accessible to Everyone. Simplified summaries of the latest research, designed for patients, caregivers and anybody who's interested.

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Regenerative Medicine Research

Browse the latest research summaries in the field of regenerative medicine for spinal cord injury patients and caregivers.

Showing 1,851-1,860 of 2,298 results

Spinal Cord InjuryRegenerative MedicineGenetics

Acidic Fibroblast Growth Factor in Spinal Cord Injury

Neurospine, 2019 • December 1, 2019

Spinal cord injury (SCI) is a devastating condition with limited regenerative capacity, leading to lifelong disability. Current treatments primarily focus on neurorehabilitation and managing complicat...

KEY FINDING: aFGF has shown promise for neural regeneration in multiple trials, including human and animal experiments, warranting further investigation as a neurotrophic factor.

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Regenerative MedicineNeurology

Do Neural Stem Cells Have a Choice? Heterogenic Outcome of Cell Fate Acquisition in Different Injury Models

International Journal of Molecular Sciences, 2019 • January 21, 2019

This review examines the behavior of transplanted neural stem cells (NSCs) in different injury models within the central nervous system (CNS). It compiles and compares various transplantation protocol...

KEY FINDING: In healthy CNS tissue, region-specific cues dominate cell fate decisions of transplanted NSCs.

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Spinal Cord InjuryRegenerative MedicineNeurology

Regenerative capacity in the lamprey spinal cord is not altered after a repeated transection

PLoS ONE, 2019 • January 30, 2019

This study investigates the resilience of spinal cord regeneration in lampreys after a second spinal transection (re-transection). The findings indicate that lampreys regenerate just as robustly after...

KEY FINDING: Lampreys exhibit normal functional recovery of swimming after two successive spinal cord transections.

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Spinal Cord InjuryRegenerative MedicineBiomedical

An anti-inflammatory peptide and brain-derived neurotrophic factor-modified hyaluronan-methylcellulose hydrogel promotes nerve regeneration in rats with spinal cord injury

International Journal of Nanomedicine, 2019 • January 1, 2019

This study investigates the efficacy of a HAMC-KAFAK/BDNF hydrogel in promoting nerve regeneration in rats with spinal cord injury. The hydrogel is designed to provide sustained release of an anti-inf...

KEY FINDING: The HAMC-KAFAK/BDNF hydrogel demonstrates minimal swelling and sustained release of KAFAK and BDNF, suggesting its suitability for localized drug delivery.

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Spinal Cord InjuryRegenerative Medicine

Use of a combination strategy to improve neuroprotection and neuroregeneration in a rat model of acute spinal cord injury

Neural Regen Res, 2019 • June 1, 2019

This study investigated the effect of combining four different strategies (INDP, DPY, FG, and MSCs) on the behavioral and morphological recovery of rats with acute SCI. The results showed that animals...

KEY FINDING: The combination therapy (DPY+ INDP + FG + MSCs) was the best strategy to promote motor and sensibility recovery in rats with acute spinal cord injury.

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Spinal Cord InjuryRegenerative MedicineNeurology

Effect of lentiviral vector-mediated overexpression of hypoxia-inducible factor 1 alpha delivered by pluronic F-127 hydrogel on brachial plexus avulsion in rats

Neural Regeneration Research, 2019 • June 1, 2019

This study investigates the effect of lentiviral vector-mediated overexpression of hypoxia-inducible factor 1 alpha (HIF-1α) delivered by pluronic F-127 hydrogel on brachial plexus avulsion in rats. T...

KEY FINDING: Delivery of HIF-1α overexpression lentiviral vectors, especially when mediated by pluronic F-127 hydrogel, effectively promotes spinal root regeneration after brachial plexus avulsion.

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Regenerative MedicineNeurologyGenetics

A Conditioning Sciatic Nerve Lesion Triggers a Pro-regenerative State in Primary Sensory Neurons Also of Dorsal Root Ganglia Non-associated With the Damaged Nerve

Front. Cell. Neurosci., 2019 • February 4, 2019

This study demonstrates that a conditioning sciatic nerve lesion induces a pro-regenerative state in primary sensory neurons of both lumbar and remote cervical DRG. The pro-regenerative state is chara...

KEY FINDING: A unilateral sciatic nerve injury leads to a bilateral increase in growth-associated protein-43 (GAP-43) and superior cervical ganglion-10 (SCG-10) mRNA and protein levels in both lumbar and cervical DRG neurons.

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Regenerative MedicineNeurologyGenetics

New perspectives on the mechanisms establishing the dorsal-ventral axis of the spinal cord

Curr Top Dev Biol, 2019 • January 1, 2019

The dorsal-ventral axis of the spinal cord is crucial for segregating neurons according to function, with motor neurons in the ventral horn and sensory interneurons in the dorsal horn. Ventral pattern...

KEY FINDING: The ventral spinal cord is patterned by Shh, which acts as a morphogen in a concentration-dependent manner.

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Regenerative MedicinePhysiologyGenetics

More Than Just a Bandage: Closing the Gap Between Injury and Appendage Regeneration

Frontiers in Physiology, 2019 • February 8, 2019

This review discusses the biophysical, biochemical, and epigenetic processes involved in regenerative healing in amphibians, particularly tail and limb regeneration in Xenopus. The review highlights t...

KEY FINDING: Reactive oxygen species (ROS) signaling is rapidly activated after injury and is required for tail regeneration. Inhibition of ROS prevents full tail regeneration and affects the activation of Wnt signaling pathways.

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Spinal Cord InjuryRegenerative MedicineNeurology

Plexina2 and CRMP2 Signaling Complex Is Activated by Nogo-A-Liganded Ngr1 to Restrict Corticospinal Axon Sprouting after Trauma

The Journal of Neuroscience, 2019 • April 24, 2019

After brain or spinal cord trauma, interaction of Nogo-A with neuronal NgR1 limits regenerative axonal sprouting and functional recovery. The Nogo-A-induced association of NgR1 with CRMP2 requires Ple...

KEY FINDING: Nogo-A induces the association of NgR1 with CRMP2, and this requires PlexinA2 as a coreceptor.

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